Back to Articles
“Resveratrol and p53: How are they involved in CRC plasticity and apoptosis?”

“Resveratrol and p53: How are they involved in CRC plasticity and apoptosis?”

#anti-cancer#anti-inflammatory#mitochondria#diabetes-metabolic#organ-protection

Summary of the article “Resveratrol and p53: How are they involved in CRC plasticity and apoptosis?”

From ScienceDirect

This article is a review explaining the role of Resveratrol (polyphenols from the skin of red grapes, mulberries, etc.) in prevention and treatment. Colorectal Cancer (CRC) with emphasis on the relationship with p53 protein, which is an important tumor suppressor, also known as the “guardian of the genome.”

The main points of the article

1. CRC is related to “Tumor Cell Plasticity”

CRC is not caused by genetics alone. but related to

lifestyle habits

chronic inflammation

epigenetic factors

DNA repair abnormalities

What is important is that cancer cells have plasticity or the ability to “Change yourself to survive,” for example.

EMT (epithelial-mesenchymal transition)

Birth of Cancer Stem Cells (CSCs)

metabolic reprogramming

The result is

Drug resistant

Metastases (metastasis)

increase the severity of the disease

2. p53 is a key regulator of cancer resistance.

p53 functions

Stop cell cycle

DNA repair

stimulate apoptosis

Reduce EMT

Reduce CSC formation

Inhibits inflammation through NF-κB

If p53 loses its function, it causes

More cancer cells survive

EMT increased

increased stemness

Metastasis occurs more easily.

3. Resveratrol helps “activate” p53.

Resveratrol is effective

anti-inflammatory

Antioxidants

Resists the division of cancer cells

reduce spread

increase apoptosis

The important mechanism is

Stimulates p53 signaling pathway

By helping

Increase p53 expression

Increase p53 stability

Reduced inhibition from MDM2

Stimulates ATM/ATR pathway

Stimulate p38/MAPK pathway

Add Bax / PUMA / Noxa

Reduce Bcl-2

cause

mitochondrial apoptosis pathway

is

Bax ↑

→ cytochrome C release ↑

→ caspase-3 activation ↑

→ cancer cell death ↑

4. Resveratrol reduces EMT and Cancer Stem Cells.

Resveratrol reduces markers of EMT such as

Snail

Slug

Vimentin

Paxillin

and add

E-cadherin

The result is

Reduce cell movement

Reduce intrusion

reduce spread

Including reducing CSC markers such as

ALDH

CD44

stemness signaling

Thus reducing the chance of recurrence and drug resistance.

5. Has the potential to be a co-therapy with chemotherapy.

The article suggests that Resveratrol may be used in combination with medications such as:

5-FU

Oxaliplatin

to help

Increase treatment efficiency

Reduce chemoresistance

Reduce side effects

increases response to medication

Brief summary

“Resveratrol uses p53 as the key.”

to

Reduce plasticity

→ Reduce EMT + Reduce CSC + Reduce metastasis

and

increase apoptosis

→ Causes cancer cells to die.

Therefore it is both a prophylactic agent (prevention).

co-therapeutic agent (additional treatment)

for CRC in the future

Summary of health business

If used with health products such as Resveratrol extracted from mulberries.

Can communicate that

“Helps support the mechanism of protection against abnormal cells through the p53 pathway.”

“Reduces inflammation and conditions that promote cancer.”

“It helps to enhance the body's natural apoptosis system.”